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The synergy between exogenous MOTS-c and exercise is not merely additive: because MOTS-c mimics one of the molecular consequences of high-intensity exercise (AMPK activation via AICAR accumulation), concurrent exercise training may amplify the peptide's effects on GLUT4 translocation and mitochondrial biogenesis
These tend to be more severe and longer-lasting in older adults
(30, 31) However, they also highlight potential cardiovascular and renal risks, emphasizing the need for careful perioperative monitoring and patient selection
Miyayama., et al., Mitochondrial electron transport is inhibited by disappearance of metallothionein in human bronchial epithelial cells following exposure to silver nitrate
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