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The reaction is initiated by a nucleophilic attack of the more N-terminal cysteine residue of the CGPC consensus active site on the target disulfide
doi: 10.3390/nu12051516 79 MatsuiSYamamotoTTakabatakeYTakahashiANamba-HamanoTMatsudaJet al
The MHRA has not licensed these drugs for neuroprotective indications, and patients should not discontinue established treatments or use GLP-1 agonists off-label for cognitive concerns without specialist neurological assessment
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