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Concomitantly, mean systolic blood pressure declines by 35 mmHg, with modest LDL improvements, providing biologically plausible support for downstream cardiovascular risk reduction
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[14] [9] Preclinical research in animal models has demonstrated that GLP-1 receptor activation may influence dopaminergic and noradrenergic neurotransmission , the same pathways targeted by conventional ADHD medications such as methylphenidate and amphetamines
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