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The first includes injectable long-acting GLP-1R agonists and the second increases the bioavailability of endogenous GLP-1 by pharmacologically inhibiting dipeptidyl peptidase-4 (DPP-4), the enzyme that removes the N-terminal amino acid and consequently inactivates GLP-1 [2]
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Human glucagon-like peptides 1 and 2 activate rat brain adenylate cyclase
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