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No significant associations were observed between treatment-related changes in neurochemical and cognitive outcomes

Gozes group has found that ADNP mutations affect MT dynamics and inhibit its interactions with Tau [348, 349], producing a tauopathy-like phenotype manifested in increased GSK-3 activity, Tau hyper-phosphorylation, and cognitive deficiency [293]

Unlike many other antioxidants that we get primarily from food, glutathione is built by our own cells to protect them from damage

Tissues can convert ALA into EPA and DHA, but because the corresponding conversions are only 1 to 10% and 0.5-5% in human tissues, this conversion process is inefficient in humans (38)