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In a 2013 study, 15 the GSH precursor N -acetyl cysteine (NAC) was administered as a single 60-min intravenous infusion (150 mg/kg) to individuals with PD and healthy controls
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Previous work has shown that the intracellular accumulation of promiscuously reactive metabolic by-products induces widespread cellular dysfunction and contributes to antibiotic lethality 2,3,4,5,6,7,8,9,10,11,12,13
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