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Cardiovasc Res 54(3):516527 Menegola E, Di Renzo F, Broccia ML, Giavini E (2006) Inhibition of histone deacetylase as a new mechanism of teratogenesis
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This beneficial effect is evident in the G protein-biased agonist Exendin-phe1, derived from a single phenylalanine substitution at position 1 in Exenatide, which displays improved receptor recycling, reduced receptor desensitization, and sustained incretin-stimulated insulin secretion ( Genetic variation at the incretin receptors The most direct answer to why people fail to respond to incretin therapies is that they might express non-functional incretin receptors
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