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Meanwhile, posttranscriptional methylation of l-Arg residues in proteins generates asymmetric dimethylarginine (ADMA), a metabolic l-Arg analog byproduct and a natural NOS inhibitor that competes with l-Arg for binding, leading to NOS uncoupling (Fig

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Recent scientific breakthroughs point to a shared underlying mechanism: profound, systemic oxidative stress and the dysregulation of the body's most critical defense molecule