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Systemic chronic low-grade inflammation and localized renal inflammatory injury play pivotal roles in the pathogenesis of obesity-related glomerulopathy (ORG), with the TXNIP/NLRP3 pathway serving as a key mediator in activating inflammatory cascades [5]
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Small peptides like GLP-1 and GLP-2 are rapidly cleared in vivo , leading to low circulating levels
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