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Under low oxygen conditions, HIF-1 acts as a key modulator of mitochondrial metabolism, actively repressing oxidative phosphorylation (OXPHOS) to reduce cellular oxygen consumption and mitochondrial-generated reactive oxygen species (ROS), which can be toxic at high levels (78)
The molecule was first isolated by Pickart in 1973 as the active component of human albumin fraction responsible for inducing older liver tissue to synthesize proteins characteristic of younger tissue an observation that led to subsequent decades of research into GHK-Cus effects on wound healing, skin regeneration, gene expression, and cellular senescence
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