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In contrast to the aforementioned findings, it was found that depletion of Gln or inhibition of glutamine metabolism in the tumor microenvironment leads to impaired T-cell function, which promotes immune escape of tumor cells and enhances cell proliferation, invasion, and metastasis
The mechanism appears to involve modulation of the trigeminal ganglion, the cluster of nerve cells that serves as the relay station for head and face pain
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Preventive treatment with liraglutide protects against development of glucose intolerance in a rat model of Wolfram syndrome
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