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Supplementation via subcutaneous or IV routes increases tissue GSH levels more effectively than oral dosing
The peptide enters intestinal epithelial cells via the PepT1 transporter and directly inhibits inflammatory signaling pathways inside the cell, including NF-kB
AT1R expression in mesangial cells and podocytes, induced by high glucose levels, promotes intracellular expression of profibrotic and pro-inflammatory mediators, such as transforming-growth factor beta (TGF- ), vascular endothelial growth factor (VGEF), and interleukin-6 (IL-6) leading to hyperplasia and hypertrophy, mainly of the proximal tubule, together with extracellular matrix production (34)
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