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This manufacturing quality variation means apparent non-response could actually reflect product problems rather than peptide ineffectiveness

However, the complexity of human brain injury and the limitations of animal models in predicting human neurological outcomes require extensive clinical validation before considering these applications

This may particularly be the case if mitochondrial dysfunction is the consequence rather than the cause of disease pathophysiology (e.g., in ATP13A2 mutation carriers with Kufor-Rakeb Syndrome

To this respect, previously published reports provide also evidences showing that collagen type I exerts indeed an inhibitory role on adipogenesis