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In metabolic dysfunctionassociated steatohepatitis (MASH), for example, it remains uncertain whether improvements in hepatic inflammation and fibrosis are exclusively secondary to weight loss and metabolic improvements, or whether direct hepatoprotective mechanisms are involved

Despite its evocative name, its precise mechanism remains incompletely understood, which is exactly what makes it an enduring subject of experimental investigation into sleep, stress and neuroprotection

The efficacy of lipoic acid, administered either intravenously or orally, in the management of neuropathic symptoms has been examined in patients with diabetes

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