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Demyelination induces axonal defects, such as nodal complex alterations, conduction blocks, and/or irreversible axon losses that clearly disrupt long-range connectivity (Nave, 2010), as in multiple sclerosis (MS), an autoimmune disease targeting OLGs

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Quantitative susceptibility mapping (QSM) studies have demonstrated that patients with T2DM and cognitive impairment exhibit heightened iron deposition in specific brain regions, including the right caudate nucleus, substantia nigra, and left putamen, compared to their cognitively intact counterparts