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This work was supported by funding from the Academy of Finland (to C.B.J., decision #336455), the Magnus Ehrnroot Foundation (to C.B.J.), the Jane and Aatos Erkko Foundation (to C.B.J., decision #230004), the Research Council Finland (to C.B.J., decision #368527
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3.3 Protein damage and the stress sensing Cells convert heat-induced protein damage into a regulated transcriptional signal through a competitive release mechanism in which the dissociation of HSF1 from inhibitory chaperone complexes serves as a central switch in the stress response (Figure 2a) (Zheng et al., 2016)
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