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In this study, we examine how diminished GPX4 function may be a critical event in triggering oxytosis/ferroptosis to perpetuate or initiate the neurodegenerative diseases and assess the possible therapeutic importance of oxytosis/ferroptosis in neurodegenerative disorders
Supplementary Table 3 (download XLSX Whole-cell proteomic analysis of Jurkat cells in response to mitochondrial GSH depletion
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