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Post-mortem studies of the autistic brain samples showed abnormal changes in the steady-state levels of complexes I-IV in the cingulate gyrus, cerebellum, thalamus, and temporal and frontal cortex [79, 80, 86, 122]
The FAK-paxillin cell signaling pathway, which governs how cells migrate to injury sites and survive there, is upregulated by BPC-157, meaning the body sends repair cells to the damaged area more efficiently and keeps them active for longer
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doi: 10.1017/S0007114517002999 256
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