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To gain insight into the cellular response to FA, we obtained human pre-B lymphoblastic leukemic Nalm6 cells deficient in the FA-metabolizing enzyme ADH5 ( ADH5 ) and explored the tolerance to FA, the accumulation of the DNA double-strand break (DSB) marker -H2AX, and the phosphorylation of the tumour suppressor p53
doi: 10.1186/s12989-018-0251-7 206
GHK-Cu (Copper Peptide) is supplied in lyophilised vial format for laboratory research applications
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