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While GHK has been shown to inhibit the inflammatory response and epithelial-mesenchymal transition (EMT) via the TGF-1/Smad 2/3 and IGF-1 pathways in bleomycin-induced fibrosis models [24], its broader anti-inflammatory effects in the context of pulmonary fibrosis pathogenesis may warrant further exploration

Overall, ferulic acid treatment confers neuroprotection against oxidative damage and neuroinflammation, improving outcomes in TBI models

It may also offer more immediate relief than glutathione injections, though glutathione injections may last longer between doses

However, randomized controlled trials are needed to determine whether antioxidant supplements slow or prevent age-related cognitive decline