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Because a fully functioning methylation cycle is required to synthesize new glutathione, the body becomes biochemically incapable of restoring its antioxidant defenses
This warning stems from preclinical studies in rodents that demonstrated dose-dependent and treatment-duration-dependent thyroid C-cell tumors at clinically relevant exposures
doi:10.1186/s40200-014-0103-4 [13] Ebner S
Given that DPP-4 clearly plays a role in the clearance of a number of hormones involved in the regulation of glucose homeostasis, the question arises of whether changes in DPP-4 activity might play a role in the pathogenesis of T2DM
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