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ROS can react with DNA and cause damage to purines and pyrimidines [6], resulting in the formation of 8-Hydroxy-deoxyguanosine (8-OHdG) [7]

Its most consistently documented mechanism involves VEGFR2 activation and Akt-eNOS signaling, promoting angiogenesis in connective tissue research models (Hsieh et al., Journal of Molecular Medicine, 2017, DOI: 10.1007/s00109-016-1488-y)

Ebenso trgt Vitamin B5 zur normalen geistigen Leistungsfhigkeit bei (EFSA)

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