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Comparative analyses between physiological testosterone replacement therapy and supraphysiological anabolic steroid abuse underscore the importance of dose and molecular structure in determining toxicity
By activating GLP-1 receptors, semaglutide enhances glucose-dependent insulin secretion, suppresses inappropriate glucagon release, and slows gastric emptying (though this effect may attenuate over time)
These issues may overlap with conditions treated through Hormone Replacement Therapy or Bioidentical hormone therapy, making professional guidance essential
[5] The most scientifically cautious interpretation is not that broad off-target inhibition has been proven, but that broader non-target effects cannot be excluded when experiments use high concentrations, long exposure windows, sensitive cell systems, or designs lacking orthogonal NNMT validation
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