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doi: 10.1038/nbt.2859 22 WangZFuWHuoMHeBLiuYTianLet al
To further investigate this, an in vitro senescence model was established using TM3 mouse Leydig cells treated with H2O2
Phenome-based approach identifies RIC1-linked Mendelian syndrome through zebrafish models, biobank associations and clinical studies
ATO treatment markedly downregulated Bcl-2, reflecting loss of mitochondrial integrity and activation of the Bax/Bakcytochrome ccaspase pathway [49, 50]
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