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Results Inhibition of glutathione metabolism depletes neuronal glutamate and affects excitatory transmission To test the hypothesis that glutathione is a significant reservoir for glutamate, we treated neuronal cells with molecular inhibitors targeting enzymes of the glutathione metabolic cycle: L-2-imidazolidone-4-carboxylate (2I4C), acivicin, L-buthionine sulfoximine (BSO), and sulforaphane

Due to its toxicity, the compound is used as an insecticide, which acts by stimulating apoptosis in nucleated cells

It is completely valid to feel overwhelmed by the sheer effort required to simply exist on high-symptom days

Multifunctional poly(amino acid) nanomedicine modulates macrophage polarization for osteosarcoma immunotherapy