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Those receiving the 25 mg and 50 mg doses of oral semaglutide were found to be more likely to reach the A1C target of less than 7 per cent, compared with those who took 14 mg
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Within synaptic vesicles, amphetamine disrupts vesicular monoamine storage through two complementary mechanisms: (A) diffusion into vesicles and proton buffering, which collapses the vesicular proton gradient required for vesicular monoamine transporter 2 (VMAT2) function
PMCID: PMC2709042
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