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The mechanism involves dampening dopamine release in reward centers like the nucleus accumbens and ventral tegmental area, which reduces the pleasurable effects of alcohol
[9] [10] Additionally, reduced food intake means less diet-induced thermogenesis, the temporary increase in metabolic rate that occurs after eating
There is likely a subset of patients, especially those such as SL, whose genome predisposes them to the development of severe refractory obesity
Patients had not plateaued
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