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showed that the blockage of CPT1A (with etomoxir) and the lipid synthesis/lipolysis (with orlistat) decreased the viability of the androgen-dependent prostate cell lines LNCaP, VCaP, and patient-derived prostate cancer cells 34
CJC-1295 was engineered with four amino acid substitutions that protect it from these enzymes, extending its functional half-life dramatically
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