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GHK-Cu has been shown in fibroblast cultures to stimulate the transcription of genes encoding for key structural proteins

Resistant cells generate more glutathione, elicit nuclear factor erythroid 2-related factor 2 (NRF2) activation, and overexpress many anti-oxidative genes such as superoxide dismutase, catalase, glutathione peroxidase, and thioredoxin reductase, providing stronger antioxidant capacity to survive in a more oxidative environment due to the sharp rise in oxidative metabolism and reactive oxygen species generation

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Prespecified subgroup, sensitivity and specificity analyses Subgroup analyses were conducted based on gender, age, body mass index (BMI), HbA1c, DM status (pre-existing or post-transplant), post-transplant eGFR, the presence of hypertension, proteinuria, heart failure, obesity, enrollment time, prior history of GLP-1RAs uses before transplant, and the concurrent use of medications, including ACEIs/ARBs, antidiabetic agents (insulin, metformin, dipeptidyl peptidase-4 inhibitors (DPP-4is), sodium-glucose cotransporter 2 inhibitors (SGLT2is), and immunosuppressants (steroids, cyclosporine and tacrolimus)