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AHK-Cu is intended strictly for research use only
(2020) BPC-157 and tendon fibroblast healing mechanisms TB-500 Studied for cell migration and cytoskeletal dynamics Interacts with actin regulation pathways Commonly referenced in wound healing models Resources & Research Studies: Goldstein et al
Foundational GHK-Cu (sister peptide) paper: stimulates collagen synthesis in human fibroblasts, effect beginning at 10^-12 to 10^-11 M and maximal at 10^-9 M the same potency window later reported for AHK-Cu, establishing the copper-tripeptide mechanism
TB-500 binds monomeric G-actin via the LKKTETQ motif, modulating the actin pool available for cytoskeletal remodeling and cell migration
