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PubMed DOI Seiwerth S, Sikiric P, Grabarevic Z, et al

Research investigations reveal that senescent cells contribute to fibrosis through SASP-mediated inflammation, and their elimination can reduce fibrotic progression, providing researchers with tools for studying senescence contributions to fibrotic diseases and developing interventions for fibrosis research models

However, this conformational change reduces the space available to accommodate the -helix of the peptide increasing the steric hindrance between the bulky ionic lock (R17 Cagri- E14 Cagri ) and ECL2