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Activation of Glp-1 receptor enhances neuronal base excision repair via Pi3k-Akt-induced expression of apurinic/apyrimidinic endonuclease 1
Cold atmospheric plasma induces the curing mechanism of diabetic wounds by regulating the oxidative stress mediators iNOS and NO, the pyroptotic mediators NLRP-3, caspase-1 and IL-1 and the angiogenesis mediators VEGF and Ang-1
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