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Interestingly, the presence of homozygosity of the UGT1A1 *28 allele (*28/*28
Key mechanisms include: Stimulation of collagen and elastin synthesis in fibroblasts Activation of lysyl oxidase for collagen cross-linking Enhanced production of glycosaminoglycans and decorin Modulation of metalloproteinase activity for controlled matrix breakdown The copper ion serves as an essential cofactor for enzymes involved in collagen synthesis and provides antioxidant protection through superoxide dismutase activation
Both P-glycoprotein and the glutathione S-conjugate carrier are expressed in the liver which is compatible with the hypothesis that these pumps could be involved in the efflux of AFB 1 and its metabolites
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