This phenomenon may be explained by carnitine influence on volatile compound behavior, particularly through its interactions with aldehydes and ketones [88]
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Notably, both systemic liraglutide and semaglutide treatments robustly activate AP/NTS, lPBN, and CeA neurons [51, 55] through GLP-1Rs expressed in glutamatergic, but not in GABAergic, neurons [55], possibly contributing to their aversive effects