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FDG-PET scans performed preoperatively and at 6 and 12 months after surgery revealed the development of a unique, treatment-dependent, polysynaptic brain circuit linking the STN to motor cortical regions, which correlated with clinical improvement in the AAV2-GAD treated patients (Niethammer et al
We found that wild-type (WT) ACTL6A and the DNBL, CC, and NBC ACTL6A mutants reversed the inhibition of cell proliferation and the reduced GCLC protein expression induced by knocking down ACTL6A expression, but the ACTL6A HR-deleted mutant did not show this restorative effect (Fig
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