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GSH protects transplanted fat grafts against ferroptosis We examined the levels of ferroptosis in the transplanted fat grafts by immunofluorescence and observed a stronger positive staining for the anti-ferroptosis markers GPX4 and SLC7A11 in the GSH-treated group than in the control group (Fig
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Although mechanisms describing the interaction between the key pathway constituents Nrf2, Keap1 and p62 are widely reviewed and discussed in literature, quantitative dynamic models bringing together these mechanisms with time-resolved data are limited
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