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To this end, Chen et al., has suggested that activation of GLP-1R with liraglutide could protect chondrocytes against endoplasmic reticulum (ER) stress, apoptosis and inflammation by decreasing the release of inflammatory mediators and regulating phosphoinositide 3-kinase (PI3K)/AKT signaling, which suggests that GLP-1R is a therapeutic target for the treatment of OA and that liraglutide could be a therapeutic candidate for clinical application [10]
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Quantification of RNA expression from ex vivo culture LMS were harvested and transferred into 2 mL tubes (P000945-LYSKO-A.0, Precellys) with ceramic beads (91-pCS-CK 28P, Peglab) containing 1 mL QIAzol Lysis Reagent (Qiagen)
The main confirmed actions were documented as: stimulating insulin secretion (incretin effect), inhibiting glucagon secretion, inhibiting gastrointestinal motility and secretion (enterogastrone / ileal brake mechanism), and regulating appetite and food intake
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