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In isolated rat aorta preparations, the peptide produced concentration-dependent vasodilation that was endothelium-dependent and abolished by L-NAME, and co-immunoprecipitation work traced the response to phosphorylation of Src, caveolin-1, and endothelial nitric oxide synthase, with reduced binding between caveolin-1 and eNOS [1]
GSH synthesized in the cytosol must be transported into the organelle, where concentration, recycling capacity and membrane transport all shape the response to stress
PMID: 162 MaashiMSAl-MualmMAl-AwsiGRLOpulenciaMJCAl-GazallyMEAbdullaevBet al
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