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Hormonal imbalance, metabolic disorders, and an increased insulin level mainly characterize the ailment
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The pathophysiological hallmark of PD is the degeneration of dopaminergic neurons in the substantia nigra (SN) 1,3,18 , leading to dysfunction within a corticobasal gangliathalamic circuit comprised of the subthalamic nucleus (STN), ventral intermediate nucleus/centromedian nucleus of the thalamus (VIM/CM), globus pallidus internus (GPi) and externus (GPe), putamen and primary motor cortex (M1) 5,19 , revealed by electrophysiological and functional/metabolic neuroimaging evidence 3,20,21,22,23
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