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The mechanism and result are similar to that of GSTP and JNK, in that GSTM1 sequesters ASK1 through complex formation and prevents its induction of the pro-apoptotic p38 and JNK portions of the MAPK signaling cascade
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For example, a study published in the Journal of Nutrition found that supplementation with N-acetylcysteine (a precursor to glutathione) eliminated negative effects of oxidative stress, even when they were exposed to high levels of oxidative stress
Furthermore, both cysteine and glycine are incorporated into the tripeptide glutathione, and thus serine is critical for both cellular redox homeostasis and xenobiotic metabolism processes [64, 65]
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