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A secondary effect of this stress is activation of Nrf2 and its target genes resulting in compensatory higher GSH levels and induction of antioxidant genes in the livers of these mice, which makes these livers more resistant to additional acute stress such as APAP toxicity [151]
Sn phm ti y c cam kt c y tem nhn, ngun gc r rng v h tr giao hng tn ni trn ton quc
[5] Apart from that, upregulation in enzymatic markers such as serum alanine aminotransferase (ALT), alkaline phosphatase (ALP), and aspartate aminotransferase (AST) is also interlinked with tissue necrosis and degeneration of hepatic tissues and, therefore, promotes hepatotoxicity. [6] As the liver plays a vital role in the metabolism, transport, and clearance of xenobiotics, it is critically susceptible to damage
Bull Exp Biol Med
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