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Despite short serum half-life, daily dosing produces robust, sustained tissue-repair effects in rodent models
Different mechanisms for -glutamyltransferase-dependent resistance to carboplatin and cisplatin
Duration: Studies have lasted up to 14 days
Furthermore, we observed that ACTL6A suppression sensitized GC cells to H 2 O 2 , erastin, and buthionine sulfoximine (BSO), which are ferroptosis inducers, but not to compounds (e.g., etoposide and doxorubicin) that inhibit cell growth through other mechanisms (Fig
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