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However, this review also critically evaluates alternative explanations for the translational gap, including limited central nervous system penetration, insufficient pharmacodynamic target engagement, a mismatch between the drugs metabolic mechanisms and the amyloid-driven pathology of established AD, and the inherent limitations of preclinical models
Achieving long-term survival in MMNST is hampered by its tendency to metastasize and recur, and its propensity to involve the spine or other structures, which preclude complete surgical resection [32]
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