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These neuroprotective effects were linked to suppression of paraquat-induced activation of the MAPK signaling cascade, a pathway known to mediate oxidative and apoptotic damage in dopaminergic systems
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While most longevity interventions target single hallmarks of aging, like NAD+ boosters for mitochondrial support [2] or senolytics to clear senescent cells [3], Epitalons appeal lies in its multi-dimensional role in aging biology
Rodrgues et al
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