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Therapy with the Stable Gastric Pentadecapeptide BPC 157

HGF/c-Met signaling is a well-established oncogenic pathway: c-Met is amplified or overexpressed in multiple cancers (gastric, lung, renal, hepatocellular) HGF/c-Met activation promotes tumor cell proliferation, survival, invasion, and metastasis c-Met inhibitors are FDA-approved cancer drugs (cabozantinib, crizotinib, capmatinib) A compound that potentiates HGF/c-Met signaling raises theoretical concerns about promoting tumor growth or transformation with chronic use

The assay's sensitivity was evaluated by determining the limits of detection (LOD) and quantification (LOQ) (Beger et al

Non-surgical and painless, with less shedding by week 4-6 and denser hair by month 3-4