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This absence of evidence deserves careful consideration because researchers have specifically looked for carcinogenic effects and found none

It modulates existing repair pathways, with outcomes determined by the quality of the signal: platelet concentration, preparation method, delivery depth, and the patients underlying biology

This indicates the existence of a GGCT-mediated Gln metabolic shunt in tumor cells: Gln is prioritized for conversion to GSH, which scavenges ROS, alleviates oxidative stress, and ultimately promotes tumor cell proliferation