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We demonstrated that, although ROS are necessary for the initiation phase of ATG, the modulation of thiols are fundamental for the progression of starvation-induced ATG ( Figure 2 )
It also modulates pineal gland function and circadian biologyrestoring nighttime melatonin release and normalizing disrupted sleepwake rhythms in older humans and animals
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Gln3 mutations dissociate responses to nitrogen limitation (nitrogen catabolite repression) and rapamycin inhibition of TorC1
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