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Understanding these variables can help clinicians and patients anticipate and manage symptoms more effectively

Wang et al, 1993), homocysteine could impair the ability of these cells to inhibit platelet aggregation, probably by reducing nitric oxide (NO) bioavailability (Stamler et al, 1993)

This system effectively alleviates hypoxia-induced mitochondrial damage, restores autophagy flow, enhances cellular stress adaptability, and ultimately reduces oxidative stress and renal tissue damage [66]