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The way it works makes sense: GLP-1 mostly changes the brainstem circuits that control feeling full, while amylin changes the area postrema and the nucleus tractus solitarius, which affects different neural paths
So the protocol sets the dose to land GHK-Cu in a sensible daily range (~1.67 mg), and the three recovery/anti-inflammatory peptides ride along at their lower 0.33 mg each
Once injected, the medication begins mimicking the natural GLP-1 hormone, which targets the hunger centers in the brain, specifically the hypothalamus
Most GLP-1 stalls last 2 to 8 weeks
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