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Atherogenicity of amino acids in the lipid-laden macrophage model system in vitro and in atherosclerotic mice: a key role for triglyceride metabolism
It is linked with increased ROS production that reduces NO bioavailability impairing endothelial function [207, 214]
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GHK-Cu inhibits its activation and thereby reduces the production of pro-inflammatory cytokines, chemokines, and adhesion molecules
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